icam-1 knock out mice Search Results


recombinant mouse icam 1  (R&D Systems)
93
R&D Systems recombinant mouse icam 1
Recombinant Mouse Icam 1, supplied by R&D Systems, used in various techniques. Bioz Stars score: 93/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Average 93 stars, based on 1 article reviews
recombinant mouse icam 1 - by Bioz Stars, 2026-03
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mouse anti-human icam-1 antibody  (Millipore)
90
Millipore mouse anti-human icam-1 antibody
Mouse Anti Human Icam 1 Antibody, supplied by Millipore, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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mouse anti icam 1  (Danaher Inc)
86
Danaher Inc mouse anti icam 1
A: Digital photograph of the LC-PMN device. See Supplemental Information for design specifications. Compartment labels indicate locations of tissues depicted in Panel B. TME = tumor microenvironment. PMN = premetastatic niche. B: Inflammatory activation visualized in 3D LSCM stacks <t>of</t> <t>ICAM-1</t> (green), UEA-1 (endothelial cells, red), and DAPI (blue) staining after 7 days of culture. Co-localization of UEA-1 and ICAM-1 indicates vascular inflammation (yellow). Inflammation of non-endothelial cells is localized by green only staining. Scale bars = 200 μm. C: ICAM-1 fluorescence intensity in regions of colocalization with UEA-1 (Vascular ICAM-1). Data is normalized as fold increase over control devices which replace with the TME with a fourth PMN compartment (no cancer in the system). * Indicates p < 0.05. D: Normalized vascular ICAM-1 in each compartment of LC-PMN devices. E: Relative non-vascular inflammation represented as a ratio of vascular to non-vascular ICAM-1 intensity discriminated based on colocalization with UEA-1. Trend lines in Panels D and E illustrate the linear gradient trends expected in a static culture system.
Mouse Anti Icam 1, supplied by Danaher Inc, used in various techniques. Bioz Stars score: 86/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/result/mouse anti icam 1/product/Danaher Inc
Average 86 stars, based on 1 article reviews
mouse anti icam 1 - by Bioz Stars, 2026-03
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48h10 anti-icam-1  (Immunotec inc)
90
Immunotec inc 48h10 anti-icam-1
A: Digital photograph of the LC-PMN device. See Supplemental Information for design specifications. Compartment labels indicate locations of tissues depicted in Panel B. TME = tumor microenvironment. PMN = premetastatic niche. B: Inflammatory activation visualized in 3D LSCM stacks <t>of</t> <t>ICAM-1</t> (green), UEA-1 (endothelial cells, red), and DAPI (blue) staining after 7 days of culture. Co-localization of UEA-1 and ICAM-1 indicates vascular inflammation (yellow). Inflammation of non-endothelial cells is localized by green only staining. Scale bars = 200 μm. C: ICAM-1 fluorescence intensity in regions of colocalization with UEA-1 (Vascular ICAM-1). Data is normalized as fold increase over control devices which replace with the TME with a fourth PMN compartment (no cancer in the system). * Indicates p < 0.05. D: Normalized vascular ICAM-1 in each compartment of LC-PMN devices. E: Relative non-vascular inflammation represented as a ratio of vascular to non-vascular ICAM-1 intensity discriminated based on colocalization with UEA-1. Trend lines in Panels D and E illustrate the linear gradient trends expected in a static culture system.
48h10 Anti Icam 1, supplied by Immunotec inc, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/result/48h10 anti-icam-1/product/Immunotec inc
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rat anti mouse icam1  (Thermo Fisher)
86
Thermo Fisher rat anti mouse icam1
A: Digital photograph of the LC-PMN device. See Supplemental Information for design specifications. Compartment labels indicate locations of tissues depicted in Panel B. TME = tumor microenvironment. PMN = premetastatic niche. B: Inflammatory activation visualized in 3D LSCM stacks <t>of</t> <t>ICAM-1</t> (green), UEA-1 (endothelial cells, red), and DAPI (blue) staining after 7 days of culture. Co-localization of UEA-1 and ICAM-1 indicates vascular inflammation (yellow). Inflammation of non-endothelial cells is localized by green only staining. Scale bars = 200 μm. C: ICAM-1 fluorescence intensity in regions of colocalization with UEA-1 (Vascular ICAM-1). Data is normalized as fold increase over control devices which replace with the TME with a fourth PMN compartment (no cancer in the system). * Indicates p < 0.05. D: Normalized vascular ICAM-1 in each compartment of LC-PMN devices. E: Relative non-vascular inflammation represented as a ratio of vascular to non-vascular ICAM-1 intensity discriminated based on colocalization with UEA-1. Trend lines in Panels D and E illustrate the linear gradient trends expected in a static culture system.
Rat Anti Mouse Icam1, supplied by Thermo Fisher, used in various techniques. Bioz Stars score: 86/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Average 86 stars, based on 1 article reviews
rat anti mouse icam1 - by Bioz Stars, 2026-03
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rat anti-mouse icam-1 mab kat-1  (Seikagaku corporation)
90
Seikagaku corporation rat anti-mouse icam-1 mab kat-1
A: Digital photograph of the LC-PMN device. See Supplemental Information for design specifications. Compartment labels indicate locations of tissues depicted in Panel B. TME = tumor microenvironment. PMN = premetastatic niche. B: Inflammatory activation visualized in 3D LSCM stacks <t>of</t> <t>ICAM-1</t> (green), UEA-1 (endothelial cells, red), and DAPI (blue) staining after 7 days of culture. Co-localization of UEA-1 and ICAM-1 indicates vascular inflammation (yellow). Inflammation of non-endothelial cells is localized by green only staining. Scale bars = 200 μm. C: ICAM-1 fluorescence intensity in regions of colocalization with UEA-1 (Vascular ICAM-1). Data is normalized as fold increase over control devices which replace with the TME with a fourth PMN compartment (no cancer in the system). * Indicates p < 0.05. D: Normalized vascular ICAM-1 in each compartment of LC-PMN devices. E: Relative non-vascular inflammation represented as a ratio of vascular to non-vascular ICAM-1 intensity discriminated based on colocalization with UEA-1. Trend lines in Panels D and E illustrate the linear gradient trends expected in a static culture system.
Rat Anti Mouse Icam 1 Mab Kat 1, supplied by Seikagaku corporation, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/result/rat anti-mouse icam-1 mab kat-1/product/Seikagaku corporation
Average 90 stars, based on 1 article reviews
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anti-human icam-1 mouse igg2a mab  (Endogen Inc)
90
Endogen Inc anti-human icam-1 mouse igg2a mab
A: Digital photograph of the LC-PMN device. See Supplemental Information for design specifications. Compartment labels indicate locations of tissues depicted in Panel B. TME = tumor microenvironment. PMN = premetastatic niche. B: Inflammatory activation visualized in 3D LSCM stacks <t>of</t> <t>ICAM-1</t> (green), UEA-1 (endothelial cells, red), and DAPI (blue) staining after 7 days of culture. Co-localization of UEA-1 and ICAM-1 indicates vascular inflammation (yellow). Inflammation of non-endothelial cells is localized by green only staining. Scale bars = 200 μm. C: ICAM-1 fluorescence intensity in regions of colocalization with UEA-1 (Vascular ICAM-1). Data is normalized as fold increase over control devices which replace with the TME with a fourth PMN compartment (no cancer in the system). * Indicates p < 0.05. D: Normalized vascular ICAM-1 in each compartment of LC-PMN devices. E: Relative non-vascular inflammation represented as a ratio of vascular to non-vascular ICAM-1 intensity discriminated based on colocalization with UEA-1. Trend lines in Panels D and E illustrate the linear gradient trends expected in a static culture system.
Anti Human Icam 1 Mouse Igg2a Mab, supplied by Endogen Inc, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Average 90 stars, based on 1 article reviews
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mouse monoclonal anti icam 1 antibody  (Santa Cruz Biotechnology)
96
Santa Cruz Biotechnology mouse monoclonal anti icam 1 antibody
A: Digital photograph of the LC-PMN device. See Supplemental Information for design specifications. Compartment labels indicate locations of tissues depicted in Panel B. TME = tumor microenvironment. PMN = premetastatic niche. B: Inflammatory activation visualized in 3D LSCM stacks <t>of</t> <t>ICAM-1</t> (green), UEA-1 (endothelial cells, red), and DAPI (blue) staining after 7 days of culture. Co-localization of UEA-1 and ICAM-1 indicates vascular inflammation (yellow). Inflammation of non-endothelial cells is localized by green only staining. Scale bars = 200 μm. C: ICAM-1 fluorescence intensity in regions of colocalization with UEA-1 (Vascular ICAM-1). Data is normalized as fold increase over control devices which replace with the TME with a fourth PMN compartment (no cancer in the system). * Indicates p < 0.05. D: Normalized vascular ICAM-1 in each compartment of LC-PMN devices. E: Relative non-vascular inflammation represented as a ratio of vascular to non-vascular ICAM-1 intensity discriminated based on colocalization with UEA-1. Trend lines in Panels D and E illustrate the linear gradient trends expected in a static culture system.
Mouse Monoclonal Anti Icam 1 Antibody, supplied by Santa Cruz Biotechnology, used in various techniques. Bioz Stars score: 96/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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mouse icam  (R&D Systems)
91
R&D Systems mouse icam
Requirement of the second Ig-like domain of TLCN for VN binding. Surface plasmon resonance analysis of protein-protein interaction between recombinant <t>ICAM</t> family molecules and VN. A, a schematic diagram shows TLCN, TLCN(1–9)/Fc, TLCN-truncated mutants TLCN(1–3)/Fc, TLCN(1–2)/Fc, and TLCN(1)/Fc, TLCN 2nd Ig-like domain deletion and substitution mutants TLCN(1,3–9)/Fc and TLCN(1,3–9)ICAM-1(2)/Fc, and two other ICAMs, ICAM-1/Fc and ICAM-2/Fc proteins. Green, red, and yellow ovals show TLCN, ICAM-1, and ICAM-2 Ig-like domains, respectively. B–I, VN was immobilized on sensor chip and assayed for binding to TLCN(1–9)/Fc (B), TLCN(1–3)/Fc (C), TLCN(1–2)/Fc (D), TLCN(1)/Fc (E), TLCN(1,3–9)/Fc (F), TLCN(1,3–9)ICAM-1(2)/Fc (G), ICAM-1/Fc (H), and ICAM-2/Fc (I) proteins at different concentrations (16.9, 33.8, 67.5, 135, and 270 nm). RU, response units.
Mouse Icam, supplied by R&D Systems, used in various techniques. Bioz Stars score: 91/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/result/mouse icam/product/R&D Systems
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mouse icam 1 fc  (R&D Systems)
95
R&D Systems mouse icam 1 fc
Requirement of the second Ig-like domain of TLCN for VN binding. Surface plasmon resonance analysis of protein-protein interaction between recombinant <t>ICAM</t> family molecules and VN. A, a schematic diagram shows TLCN, TLCN(1–9)/Fc, TLCN-truncated mutants TLCN(1–3)/Fc, TLCN(1–2)/Fc, and TLCN(1)/Fc, TLCN 2nd Ig-like domain deletion and substitution mutants TLCN(1,3–9)/Fc and TLCN(1,3–9)ICAM-1(2)/Fc, and two other ICAMs, ICAM-1/Fc and ICAM-2/Fc proteins. Green, red, and yellow ovals show TLCN, ICAM-1, and ICAM-2 Ig-like domains, respectively. B–I, VN was immobilized on sensor chip and assayed for binding to TLCN(1–9)/Fc (B), TLCN(1–3)/Fc (C), TLCN(1–2)/Fc (D), TLCN(1)/Fc (E), TLCN(1,3–9)/Fc (F), TLCN(1,3–9)ICAM-1(2)/Fc (G), ICAM-1/Fc (H), and ICAM-2/Fc (I) proteins at different concentrations (16.9, 33.8, 67.5, 135, and 270 nm). RU, response units.
Mouse Icam 1 Fc, supplied by R&D Systems, used in various techniques. Bioz Stars score: 95/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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mouse anti rat icam 1  (Bio-Rad)
93
Bio-Rad mouse anti rat icam 1
Requirement of the second Ig-like domain of TLCN for VN binding. Surface plasmon resonance analysis of protein-protein interaction between recombinant <t>ICAM</t> family molecules and VN. A, a schematic diagram shows TLCN, TLCN(1–9)/Fc, TLCN-truncated mutants TLCN(1–3)/Fc, TLCN(1–2)/Fc, and TLCN(1)/Fc, TLCN 2nd Ig-like domain deletion and substitution mutants TLCN(1,3–9)/Fc and TLCN(1,3–9)ICAM-1(2)/Fc, and two other ICAMs, ICAM-1/Fc and ICAM-2/Fc proteins. Green, red, and yellow ovals show TLCN, ICAM-1, and ICAM-2 Ig-like domains, respectively. B–I, VN was immobilized on sensor chip and assayed for binding to TLCN(1–9)/Fc (B), TLCN(1–3)/Fc (C), TLCN(1–2)/Fc (D), TLCN(1)/Fc (E), TLCN(1,3–9)/Fc (F), TLCN(1,3–9)ICAM-1(2)/Fc (G), ICAM-1/Fc (H), and ICAM-2/Fc (I) proteins at different concentrations (16.9, 33.8, 67.5, 135, and 270 nm). RU, response units.
Mouse Anti Rat Icam 1, supplied by Bio-Rad, used in various techniques. Bioz Stars score: 93/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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p14 cd45.1 +/+ icam1 −/− h2b bm1 mice  (Jackson Laboratory)
90
Jackson Laboratory p14 cd45.1 +/+ icam1 −/− h2b bm1 mice
Requirement of the second Ig-like domain of TLCN for VN binding. Surface plasmon resonance analysis of protein-protein interaction between recombinant <t>ICAM</t> family molecules and VN. A, a schematic diagram shows TLCN, TLCN(1–9)/Fc, TLCN-truncated mutants TLCN(1–3)/Fc, TLCN(1–2)/Fc, and TLCN(1)/Fc, TLCN 2nd Ig-like domain deletion and substitution mutants TLCN(1,3–9)/Fc and TLCN(1,3–9)ICAM-1(2)/Fc, and two other ICAMs, ICAM-1/Fc and ICAM-2/Fc proteins. Green, red, and yellow ovals show TLCN, ICAM-1, and ICAM-2 Ig-like domains, respectively. B–I, VN was immobilized on sensor chip and assayed for binding to TLCN(1–9)/Fc (B), TLCN(1–3)/Fc (C), TLCN(1–2)/Fc (D), TLCN(1)/Fc (E), TLCN(1,3–9)/Fc (F), TLCN(1,3–9)ICAM-1(2)/Fc (G), ICAM-1/Fc (H), and ICAM-2/Fc (I) proteins at different concentrations (16.9, 33.8, 67.5, 135, and 270 nm). RU, response units.
P14 Cd45.1 +/+ Icam1 −/− H2b Bm1 Mice, supplied by Jackson Laboratory, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Image Search Results


A: Digital photograph of the LC-PMN device. See Supplemental Information for design specifications. Compartment labels indicate locations of tissues depicted in Panel B. TME = tumor microenvironment. PMN = premetastatic niche. B: Inflammatory activation visualized in 3D LSCM stacks of ICAM-1 (green), UEA-1 (endothelial cells, red), and DAPI (blue) staining after 7 days of culture. Co-localization of UEA-1 and ICAM-1 indicates vascular inflammation (yellow). Inflammation of non-endothelial cells is localized by green only staining. Scale bars = 200 μm. C: ICAM-1 fluorescence intensity in regions of colocalization with UEA-1 (Vascular ICAM-1). Data is normalized as fold increase over control devices which replace with the TME with a fourth PMN compartment (no cancer in the system). * Indicates p < 0.05. D: Normalized vascular ICAM-1 in each compartment of LC-PMN devices. E: Relative non-vascular inflammation represented as a ratio of vascular to non-vascular ICAM-1 intensity discriminated based on colocalization with UEA-1. Trend lines in Panels D and E illustrate the linear gradient trends expected in a static culture system.

Journal: bioRxiv

Article Title: Adaptable Fabrication of Vascularized Milliscale Tissues in Membrane-Free Organ Chips Manufactured with 3D Printed Molds

doi: 10.1101/2023.12.06.570409

Figure Lengend Snippet: A: Digital photograph of the LC-PMN device. See Supplemental Information for design specifications. Compartment labels indicate locations of tissues depicted in Panel B. TME = tumor microenvironment. PMN = premetastatic niche. B: Inflammatory activation visualized in 3D LSCM stacks of ICAM-1 (green), UEA-1 (endothelial cells, red), and DAPI (blue) staining after 7 days of culture. Co-localization of UEA-1 and ICAM-1 indicates vascular inflammation (yellow). Inflammation of non-endothelial cells is localized by green only staining. Scale bars = 200 μm. C: ICAM-1 fluorescence intensity in regions of colocalization with UEA-1 (Vascular ICAM-1). Data is normalized as fold increase over control devices which replace with the TME with a fourth PMN compartment (no cancer in the system). * Indicates p < 0.05. D: Normalized vascular ICAM-1 in each compartment of LC-PMN devices. E: Relative non-vascular inflammation represented as a ratio of vascular to non-vascular ICAM-1 intensity discriminated based on colocalization with UEA-1. Trend lines in Panels D and E illustrate the linear gradient trends expected in a static culture system.

Article Snippet: Rabbit anti-Ki67 (Abcam, ab15580), Mouse anti-ICAM-1 (Abcam, ab171123), or Rabbit anti-laminin (Abcam, ab11575) were diluted 1:100 in 1% BSA with 0.1% Triton-X.

Techniques: Activation Assay, Staining, Fluorescence

Requirement of the second Ig-like domain of TLCN for VN binding. Surface plasmon resonance analysis of protein-protein interaction between recombinant ICAM family molecules and VN. A, a schematic diagram shows TLCN, TLCN(1–9)/Fc, TLCN-truncated mutants TLCN(1–3)/Fc, TLCN(1–2)/Fc, and TLCN(1)/Fc, TLCN 2nd Ig-like domain deletion and substitution mutants TLCN(1,3–9)/Fc and TLCN(1,3–9)ICAM-1(2)/Fc, and two other ICAMs, ICAM-1/Fc and ICAM-2/Fc proteins. Green, red, and yellow ovals show TLCN, ICAM-1, and ICAM-2 Ig-like domains, respectively. B–I, VN was immobilized on sensor chip and assayed for binding to TLCN(1–9)/Fc (B), TLCN(1–3)/Fc (C), TLCN(1–2)/Fc (D), TLCN(1)/Fc (E), TLCN(1,3–9)/Fc (F), TLCN(1,3–9)ICAM-1(2)/Fc (G), ICAM-1/Fc (H), and ICAM-2/Fc (I) proteins at different concentrations (16.9, 33.8, 67.5, 135, and 270 nm). RU, response units.

Journal: The Journal of Biological Chemistry

Article Title: Vitronectin Induces Phosphorylation of Ezrin/Radixin/Moesin Actin-binding Proteins through Binding to Its Novel Neuronal Receptor Telencephalin *

doi: 10.1074/jbc.M112.383851

Figure Lengend Snippet: Requirement of the second Ig-like domain of TLCN for VN binding. Surface plasmon resonance analysis of protein-protein interaction between recombinant ICAM family molecules and VN. A, a schematic diagram shows TLCN, TLCN(1–9)/Fc, TLCN-truncated mutants TLCN(1–3)/Fc, TLCN(1–2)/Fc, and TLCN(1)/Fc, TLCN 2nd Ig-like domain deletion and substitution mutants TLCN(1,3–9)/Fc and TLCN(1,3–9)ICAM-1(2)/Fc, and two other ICAMs, ICAM-1/Fc and ICAM-2/Fc proteins. Green, red, and yellow ovals show TLCN, ICAM-1, and ICAM-2 Ig-like domains, respectively. B–I, VN was immobilized on sensor chip and assayed for binding to TLCN(1–9)/Fc (B), TLCN(1–3)/Fc (C), TLCN(1–2)/Fc (D), TLCN(1)/Fc (E), TLCN(1,3–9)/Fc (F), TLCN(1,3–9)ICAM-1(2)/Fc (G), ICAM-1/Fc (H), and ICAM-2/Fc (I) proteins at different concentrations (16.9, 33.8, 67.5, 135, and 270 nm). RU, response units.

Article Snippet: Mouse TLCN(1–9)/Fc (mouse ICAM-5/Fc), mouse ICAM-1/Fc, and mouse ICAM-2/Fc were purchased from R&D Systems.

Techniques: Binding Assay, SPR Assay, Recombinant